Please use this identifier to cite or link to this item: https://repository.monashhealth.org/monashhealthjspui/handle/1/41824
Title: Four pediatric patients with autosomal recessive polycystic kidney disease developed new-onset diabetes after renal transplantation.
Authors: Carter S.A.;Kitching A.R. ;Johnstone L.M.
Institution: (Carter, Kitching, Johnstone) Department of Nephrology, Monash Children's Hospital, Monash Health, 246 Clayton Road, Clayton, VIC 3168, Australia (Kitching) Centre for Inflammatory Diseases, Department of Medicine, Monash University, Melbourne, VIC, Australia
Issue Date: 13-Apr-2015
Copyright year: 2014
Publisher: Blackwell Publishing Inc. (E-mail: subscrip@blackwellpub.com)
Place of publication: United States
Publication information: Pediatric Transplantation. 18 (7) (pp 698-705), 2014. Date of Publication: 2014.
Journal: Pediatric Transplantation
Abstract: NODAT is increasingly prevalent. Compared with adult recipients, NODAT is less prevalent in pediatric renal transplant recipients; however, some risk factors for its development in young patients have been defined. We report four pediatric renal transplant recipients with ARPKD who developed NODAT. We review the current pediatric NODAT literature and hypothesize that ARPKD may be an additional risk factor for NODAT.Copyright © 2014 John Wiley & Sons A/S. Published by John Wiley & Sons Ltd.
DOI: http://monash.idm.oclc.org/login?url=http://dx.doi.org/10.1111/petr.12332
PubMed URL: 25118046 [http://www.ncbi.nlm.nih.gov/pubmed/?term=25118046]
ISSN: 1397-3142
URI: https://repository.monashhealth.org/monashhealthjspui/handle/1/41824
Type: Article
Type of Clinical Study or Trial: Case series or case report
Appears in Collections:Articles

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