Please use this identifier to cite or link to this item: https://repository.monashhealth.org/monashhealthjspui/handle/1/37628
Title: Editorial: Advances in mechanisms of renal fibrosis.
Authors: Lan H.Y.;Nikolic-Paterson D.J. 
Institution: (Lan) Department of Medicine and Therapeutics, Li Ka Shing Institute of Health Sciences, Chinese University of Hong Kong, Hong Kong (Nikolic-Paterson) Department of Nephrology, Monash Medical Centre, Monash University Centre for Inflammatory Diseases, Monash Health, Clayton, VIC, Australia
Issue Date: 14-Apr-2018
Copyright year: 2018
Publisher: Frontiers Media S.A. (E-mail: info@frontiersin.org)
Place of publication: Switzerland
Publication information: Frontiers in Physiology. 9 (MAR) (no pagination), 2018. Article Number: 284. Date of Publication: 23 Mar 2018.
Journal: Frontiers in Physiology
DOI: http://monash.idm.oclc.org/login?url=http://dx.doi.org/10.3389/fphys.2018.00284
Link to associated publication: Click here for full text options
ISSN: 1664-042X (electronic)
URI: https://repository.monashhealth.org/monashhealthjspui/handle/1/37628
Type: Editorial
Subjects: renin angiotensin aldosterone system
*TGF beta signaling
upregulation
angiotensin converting enzyme 2/ec [Endogenous Compound]
angiotensin II/ec [Endogenous Compound]
angiotensin[1-7]/ec [Endogenous Compound]
CXCL16 chemokine/ec [Endogenous Compound]
histone deacetylase/ec [Endogenous Compound]
homeodomain interacting protein kinase 2/ec [Endogenous Compound]
Janus kinase 3/ec [Endogenous Compound]
microRNA 200/ec [Endogenous Compound]
microRNA 21/ec [Endogenous Compound]
monocyte chemotactic protein 1/ec [Endogenous Compound]
osteogenic protein 1/ec [Endogenous Compound]
secondary lymphoid tissue chemokine/ec [Endogenous Compound]
*Smad2 protein/ec [Endogenous Compound]
*Smad3 protein/ec [Endogenous Compound]
*Smad4 protein/ec [Endogenous Compound]
*Smad7 protein/ec [Endogenous Compound]
STAT6 protein/ec [Endogenous Compound]
stress activated protein kinase/ec [Endogenous Compound]
*transforming growth factor beta1/ec [Endogenous Compound]
*kidney fibrosis/et [Etiology]
apoptosis
cytokine production
disease course
down regulation
editorial
enzyme activation
enzyme inhibition
nonhuman
protein expression
*protein function
protein phosphorylation
protein RNA binding
*protein function
protein phosphorylation
protein RNA binding
renin angiotensin aldosterone system
*TGF beta signaling
upregulation
disease course
apoptosis
cytokine production
down regulation
Editorial
enzyme activation
enzyme inhibition
*kidney fibrosis / *etiology
nonhuman
protein expression
Type of Clinical Study or Trial: Opinion, perspective or news
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